[Federal Register Volume 73, Number 129 (Thursday, July 3, 2008)]
[Notices]
[Page 38233]
From the Federal Register Online via the Government Publishing Office [www.gpo.gov]
[FR Doc No: E8-15182]
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DEPARTMENT OF HEALTH AND HUMAN SERVICES
National Institutes of Health
Public Teleconference Regarding Licensing and Collaborative
Research Opportunities for: Methods and Compositions Relating to
Detecting Dihydropyrimidine Dehydrogenase (DPD)
AGENCY: National Institutes of Health, Public Health Service, HHS.
ACTION: Notice.
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Technology Summary
This technology relates to a method of detecting DPD Splicing
Mutations.
Technology Description
Scientists at the National Cancer Institute have discovered a
method detecting DPD Splicing Mutations. This method can identify
patients with such mutations, and thereby alert the health care
provider that the patient will have an adverse reaction to the
chemotherapeutic agent, 5-Fluorouracil.
The invention relates to methods and compositions that are useful
for detecting deficiencies in DPD levels in mammals including humans.
Cancer patients having a DPD deficiency are at risk of a severe toxic
reaction to the commonly used anticancer agent 5-fluorouracil (5-FU).
The technology encompasses DPD genes from human and pig, methods for
detecting the level of nucleic acids that encode DPD in a patient, and
nucleic acids that are useful as probes for this purpose.
Novel applications of the methods include:
Screening of patients prior to the administration of the
chemotherapeutic agent, 5-Fluorouracil.
Diminishing and potentially eliminating the severe side
effects of 5-Fluorauracil in patients.
Competitive Advantage of Our Technology
5-Fluorouracil (5-FU) is a therapeutic for the treatment of
multiple cancers, including breast and colon cancers. In the United
States, approximately 275,000 cancer patients receive 5-FU annually. It
is estimated that three percent (3%) of those patients develop some
degree of toxic reaction. Patients suffering toxic reactions are
difficult and expensive to treat further. Approximately, 15% of those
developing toxic reaction, will die as a result of exposure to 5-FU.
Death is typically caused by cardiotoxicity. More than 1,300 patients
in the United States die each year as a result of 5-FU toxicity. These
deaths are all potentially avoidable if patients that are likely to get
adverse reaction with 5-FU treatment are detected prior to treatment.
Patent Estate
This technology consists of the following patents and patent
applications:
I. United States Patent Number 5,856,454 entitled ``cDNA for Human
and Pig Dihydropyrimidine Dehydrogenase,'' issued January 5, 1999 (HHS
Ref. No. E-157-1994/0-US-01);
II. United States Patent Number 6,015,673 entitled ``Cloning and
Expression of cDNA for Human Dihydropyrimidine Dehydrogenase,'' issued
January 18, 2000 (HHS Ref. No. E-157-1994/0-US-03);
III. United States Patent Number 6,787,306 entitled ``Methods and
Compositions for Detecting Dihydropyrimidine Dehydrogenase Splicing
Mutations,'' issued September 7, 2004 (HHS Ref. No. E-157-1994/1-US-
01);
IV. United States Pre-Grant Publication number 2005/0136433A1
corresponding to application serial number 10/911237 entitled ``Methods
and Compositions for Detecting Dihydropyrimidine Dehydrogenase Splicing
Mutations,'' published June 23, 2005 (HHS Ref. No. E-157-1994/1-US-19)
and all issued and pending counterparts in Europe, Canada, and
Australia.
Next Step: Teleconference
There will be a teleconference where the principal investigator
will explain this technology. Licensing and collaborative research
opportunities will also be discussed. If you are interested in
participating in this teleconference please call or e-mail Mojdeh
Bahar; (301) 435-2950; [email protected]. OTT will then e-mail you
the date, time and number for the teleconference.
Dated: June 26, 2008.
Richard U. Rodriguez,
Director, Division of Technology Development and Transfer, Office of
Technology Transfer. National Institutes of Health.
[FR Doc. E8-15182 Filed 7-2-08; 8:45 am]
BILLING CODE 4140-01-P