[Federal Register Volume 69, Number 149 (Wednesday, August 4, 2004)]
[Notices]
[Pages 47145-47149]
From the Federal Register Online via the Government Publishing Office [www.gpo.gov]
[FR Doc No: 04-17507]


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ENVIRONMENTAL PROTECTION AGENCY

[OPP-2004-0222; FRL-7369-6]


Acetamiprid; Notice of Filing a Pesticide Petition to Establish a 
Tolerance for a Certain Pesticide Chemical in or on Food

AGENCY: Environmental Protection Agency (EPA).

ACTION: Notice.

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SUMMARY: This notice announces the initial filing of a pesticide 
petition proposing the establishment of regulations for residues of a 
certain pesticide chemical in or on various food commodities.

DATES: Comments, identified by docket identification (ID) number OPP-
2004-0222, must be received on or before September 3, 2004.

ADDRESSES:  Comments may be submitted electronically, by mail, or 
through hand delivery/courier. Follow the detailed instructions as 
provided in Unit I. of the SUPPLEMENTARY INFORMATION.

FOR FURTHER INFORMATION CONTACT:  Akiva Abramovitch, Registration 
Division (7505C), Office of Pesticide Programs, Environmental 
Protection Agency, 1200 Pennsylvania Ave., NW., Washington, DC 20460-
0001; telephone number: (703) 308-8328; e-mail address: 
[email protected].

SUPPLEMENTARY INFORMATION:

I. General Information

A. Does this Action Apply to Me?

     Potentially affected entities may include, but are not limited to:
     Industry, (NAICS 111)
     Crop production (NAICS 1112)
     Animal production, (NAICS 311)
     Food manufacturing, (NAICS 32532)
     This listing is not intended to be exhaustive, but rather provides 
a guide for readers regarding entities likely to be affected by this 
action. Other types of entities not listed in this unit could also be 
affected. The North American Industrial Classification System (NAICS) 
codes have been provided to assist you and others in determining 
whether this action might apply to certain entities. If you have any 
questions regarding the applicability of this action to a particular 
entity, consult the person listed under FOR FURTHER INFORMATION 
CONTACT.

[[Page 47146]]

B. How Can I Get Copies of this Document and Other Related Information?

    1. Docket. EPA has established an official public docket for this 
action under docket ID number OPP-2004-0222. The official public docket 
consists of the documents specifically referenced in this action, any 
public comments received, and other information related to this action. 
Although, a part of the official docket, the public docket does not 
include Confidential Business Information (CBI) or other information 
whose disclosure is restricted by statute. The official public docket 
is the collection of materials that is available for public viewing at 
the Public Information and Records Integrity Branch (PIRIB), Rm. 119, 
Crystal Mall 2, 1801 South Bell St., Arlington, VA. This 
docket facility is open from 8:30 a.m. to 4 p.m., Monday through 
Friday, excluding legal holidays. The docket telephone number is (703) 
305-5805.
    2. Electronic access. You may access this Federal Register document 
electronically through the EPA Internet under the ``Federal Register'' 
listings at http://www.epa.gov/fedrgstr/.
     An electronic version of the public docket is available through 
EPA's electronic public docket and comment system, EPA Dockets. You may 
use EPA Dockets at http://www.epa.gov/edocket/ to submit or view public 
comments, access the index listing of the contents of the official 
public docket, and to access those documents in the public docket that 
are available electronically. Although, not all docket materials may be 
available electronically, you may still access any of the publicly 
available docket materials through the docket facility identified in 
Unit I.B.1. Once in the system, select ``search,'' then key in the 
appropriate docket ID number.
     Certain types of information will not be placed in the EPA 
Dockets. Information claimed as CBI and other information whose 
disclosure is restricted by statute, which is not included in the 
official public docket, will not be available for public viewing in 
EPA's electronic public docket. EPA's policy is that copyrighted 
material will not be placed in EPA's electronic public docket but will 
be available only in printed, paper form in the official public docket. 
To the extent feasible, publicly available docket materials will be 
made available in EPA's electronic public docket. When a document is 
selected from the index list in EPA Dockets, the system will identify 
whether the document is available for viewing in EPA's electronic 
public docket. Although, not all docket materials may be available 
electronically, you may still access any of the publicly available 
docket materials through the docket facility identified in Unit I.B. 
EPA intends to work towards providing electronic access to all of the 
publicly available docket materials through EPA's electronic public 
docket.
     For public commenters, it is important to note that EPA's policy 
is that public comments, whether submitted electronically or on paper, 
will be made available for public viewing in EPA's electronic public 
docket as EPA receives them and without change, unless the comment 
contains copyrighted material, CBI, or other information whose 
disclosure is restricted by statute. When EPA identifies a comment 
containing copyrighted material, EPA will provide a reference to that 
material in the version of the comment that is placed in EPA's 
electronic public docket. The entire printed comment, including the 
copyrighted material, will be available in the public docket.
     Public comments submitted on computer disks that are mailed or 
delivered to the docket will be transferred to EPA's electronic public 
docket. Public comments that are mailed or delivered to the docket will 
be scanned and placed in EPA's electronic public docket. Where 
practical, physical objects will be photographed, and the photograph 
will be placed in EPA's electronic public docket along with a brief 
description written by the docket staff.

C. How and to Whom Do I Submit Comments?

     You may submit comments electronically, by mail, or through hand 
delivery/courier. To ensure proper receipt by EPA, identify the 
appropriate docket ID number in the subject line on the first page of 
your comment. Please ensure that your comments are submitted within the 
specified comment period. Comments received after the close of the 
comment period will be marked ``late.'' EPA is not required to consider 
these late comments. If you wish to submit CBI or information that is 
otherwise protected by statute, please follow the instructions in Unit 
I.D. Do not use EPA Dockets or e-mail to submit CBI or information 
protected by statute.
    1. Electronically. If you submit an electronic comment as 
prescribed in this unit, EPA recommends that you include your name, 
mailing address, and an e-mail address or other contact information in 
the body of your comment. Also, include this contact information on the 
outside of any disk or CD ROM you submit, and in any cover letter 
accompanying the disk or CD ROM. This ensures that you can be 
identified as the submitter of the comment and allows EPA to contact 
you in case EPA cannot read your comment due to technical difficulties 
or needs further information on the substance of your comment. EPA's 
policy is that EPA will not edit your comment, and any identifying or 
contact information provided in the body of a comment will be included 
as part of the comment that is placed in the official public docket, 
and made available in EPA's electronic public docket. If EPA cannot 
read your comment due to technical difficulties and cannot contact you 
for clarification, EPA may not be able to consider your comment.
    i. EPA Dockets. Your use of EPA's electronic public docket to 
submit comments to EPA electronically is EPA's preferred method for 
receiving comments. Go directly to EPA Dockets at http://www.epa.gov/edocket/, and follow the online instructions for submitting comments. 
Once in the system, select ``search,'' and then key in docket ID number 
OPP-2004-0222. The system is an ``anonymous access'' system, which 
means EPA will not know your identity, e-mail address, or other contact 
information unless you provide it in the body of your comment.
    ii. E-mail. Comments may be sent by e-mail to [email protected], 
Attention: Docket ID number OPP-2004-0222. In contrast to EPA's 
electronic public docket, EPA's e-mail system is not an ``anonymous 
access'' system. If you send an e-mail comment directly to the docket 
without going through EPA's electronic public docket, EPA's e-mail 
system automatically captures your e-mail address. E-mail addresses 
that are automatically captured by EPA's e-mail system are included as 
part of the comment that is placed in the official public docket, and 
made available in EPA's electronic public docket.
    iii. Disk or CD ROM. You may submit comments on a disk or CD ROM 
that you mail to the mailing address identified in Unit I.C.2. These 
electronic submissions will be accepted in WordPerfect or ASCII file 
format. Avoid the use of special characters and any form of encryption.
    2. By mail. Send your comments to: Public Information and Records 
Integrity Branch (PIRIB) (7502C), Office of Pesticide Programs (OPP), 
Environmental Protection Agency, 1200 Pennsylvania Ave., NW., 
Washington, DC 20460-0001, Attention: Docket ID number OPP-2004-0222.
    3. By hand delivery or courier. Deliver your comments to: Public 
Information

[[Page 47147]]

and Records Integrity Branch (PIRIB), Office of Pesticide Programs 
(OPP), Environmental Protection Agency, Rm. 119, Crystal Mall 
2, 1801 South Bell St., Arlington, VA, Attention: Docket ID 
number OPP-2004-0222. Such deliveries are only accepted during the 
docket's normal hours of operation as identified in Unit I.B.1.

D. How Should I Submit CBI to the Agency?

     Do not submit information that you consider to be CBI 
electronically through EPA's electronic public docket or by e-mail. You 
may claim information that you submit to EPA as CBI by marking any part 
or all of that information as CBI (if you submit CBI on disk or CD ROM, 
mark the outside of the disk or CD ROM as CBI and then identify 
electronically within the disk or CD ROM the specific information that 
is CBI). Information so marked will not be disclosed except in 
accordance with procedures set forth in 40 CFR part 2.
     In addition to one complete version of the comment that includes 
any information claimed as CBI, a copy of the comment that does not 
contain the information claimed as CBI must be submitted for inclusion 
in the public docket and EPA's electronic public docket. If you submit 
the copy that does not contain CBI on disk or CD ROM, mark the outside 
of the disk or CD ROM clearly that it does not contain CBI. Information 
not marked as CBI will be included in the public docket and EPA's 
electronic public docket without prior notice. If you have any 
questions about CBI or the procedures for claiming CBI, please consult 
the person listed under FOR FURTHER INFORMATION CONTACT.

E. What Should I Consider as I Prepare My Comments for EPA?

     You may find the following suggestions helpful for preparing your 
comments:
    1. Explain your views as clearly as possible.
    2. Describe any assumptions that you used.
    3. Provide copies of any technical information and/or data you used 
that support your views.
    4. If you estimate potential burden or costs, explain how you 
arrived at the estimate that you provide.
    5. Provide specific examples to illustrate your concerns.
    6. Make sure to submit your comments by the deadline in this 
notice.
    7. To ensure proper receipt by EPA, be sure to identify the docket 
ID number assigned to this action in the subject line on the first page 
of your response. You may also, provide the name, date, and Federal 
Register citation.

II. What Action is the Agency Taking?

     EPA has received a pesticide petition as follows proposing the 
establishment and/or amendment of regulations for residues of a certain 
pesticide chemical in or on various food commodities under section 408 
of the Federal Food, Drug, and Cosmetic Act (FFDCA), 21 U.S.C. 346a. 
EPA has determined that this petition contains data or information 
regarding the elements set forth in FFDCA section 408(d)(2); however, 
EPA has not fully evaluated the sufficiency of the submitted data at 
this time or whether the data support granting of the petition. 
Additional data may be needed before EPA rules on the petition.

List of Subjects

     Environmental protection, Agricultural commodities, Feed 
additives, Food additives, Pesticides and pests, Reporting and 
recordkeeping requirements.

    Dated: July 16, 2004.
Lois Rossi,
Director, Registration Division, Office of Pesticide Programs.

Summary of Petition

     The petitioner's summary of the pesticide petition is printed 
below as required by FFDCA section 408(d)(3). The summary of the 
petition was prepared by Nippon Soda Company, Ltd. % Nisson America 
Inc., and represents the view of the petitioner. The petition summary 
announces the availability of a description of the analytical methods 
available to EPA for the detection and measurement of the pesticide 
chemical residues or an explanation of why no such method is needed.

 Nippon Soda Company, Ltd.

 PP 3F6575

     EPA has received a pesticide petition (PP 3F6575) from Nippon Soda 
Co., Ltd., c/o Nisso America Inc., 220 East 42nd Street, Suite 3002, 
New York, NY, 10017. This petition proposes, pursuant to section 408(d) 
of the Federal Food, Drug and Cosmetic Act (FFDCA), 21 U.S.C. 346a(d), 
to amend 40 CFR 180.578, by establishing tolerances for the residues of 
acetamiprid in tuberous and corm vegetables as given below. The 
proposed analytical method is by LC/MS/MS. Pursuant to section 
408(d)(2) of the FFDCA, as amended by the Food Quality Protection Act 
(FQPA), Nippon Soda Co., Ltd. has submitted the following summary of 
information, data and rationales in support of their pesticide petition 
and authorization for the summary to be published in the Federal 
Register in a notice of receipt of the petition. This summary was 
prepared by Nippon Soda Co., Ltd. EPA is in the process of evaluating 
the petition and has not determined whether the data supports granting 
of the petition. EPA may have made minor edits to the summary for the 
purpose of clarity.

A. Residue Chemistry

    1. Plant metabolism. The metabolism of acetamiprid in plants is 
well understood, having been investigated in eggplant, apples, cabbage, 
carrots, and cotton. Metabolism in plants primarily involves 
demethylation of the N-methyl group with subsequent hydrolysis of the 
acetamidine function to give the N-acetyl compound. This compound is 
then hydrolyzed to the corresponding amine followed by oxidation to the 
alcohol and acid. Conjugation of the alcohol with glucose is also 
significant. Degradation of the side chain without loss of the N-methyl 
group is seen in carrots since this is the major metabolic route in 
soil.
    2. Analytical method. Based upon the metabolism of acetamiprid in 
plants and the toxicology of the parent and metabolites, quantification 
of the parent acetamiprid is sufficient to determine toxic residues. As 
a result a method has been developed which involves extraction of 
acetamiprid from crops with methanol, filtration, partitioning and 
cleanup, and analysis by LC/MS/MS methods. The limit of quantification 
(LOQ) for the method is 0.01 parts per million (ppm) and the method 
detection limit (MDL) is 0.0003 ppm.
    3. Magnitude of residues. Magnitude of residue studies were 
conducted in potatoes as the representative crop for tuberous and corm 
vegetables. Trials were conducted in all of the major use areas for 
each of the crops as specified in the Residue Chemistry Guidelines 
OPPTS 860.1500 with applications at the maximum label use rate for each 
crop. As a result of the field trials the following tolerances are 
proposed for each of the tuberous and corm crop groups: 0.01 ppm. A 
processing study was also conducted with potatoes however even at 5X 
the labeled rate, acetamiprid residues were below the LOQ in the raw 
agricultural commodity and collected potato processing fractions were 
not analyzed.

B. Toxicological Profile

    1. Acute toxicity for technical acetamiprid. The acute oral LD-
50 for acetamiprid was 146 milligrams/

[[Page 47148]]

kilogram (mg/kg) for female Sprague-Dawley rats and 217 for male rats. 
The acute dermal LD-50 for acetamiprid was greater than 
2,000 mg/kg in rats. The acute 4 hour inhalation LC-50 for 
acetamiprid was greater than 1.15 milligrams/Liter (mg/L), the highest 
attainable concentration. Acetamiprid was not irritating to the eyes or 
skin and was not considered to be a sensitizing agent. The no observed 
effect level (NOEL) for acute neurotoxicity was 10 gram/kilogram (g/kg) 
and no evidence of neuropathy was noted.
    Acute toxicity for formulated acetamiprid 70WP. The acute oral LD-
50 for Acetamiprid 70WP was 944 mg/kg for female Sprague-
Dawley rats and 1,107 mg/kg for male rats. The acute dermal LD-
50 for formulated acetamiprid was greater than 2,000 mg/kg 
in rats. The acute inhalation LC-50 (4 hours) for 
acetamiprid 70WP was determined to be greater than 2.88 milligrams per 
Liter (mg/L), the highest attainable concentration. Acetamiprid 70WP 
was concluded to be a mild eye irritant and slight skin irritant. There 
were no indications of skin sensitization for the formulated product.
    2. Genoxicity for technical acetamiprid. Based on the weight of the 
evidence provided by a complete test battery, acetamiprid is neither 
mutagenic nor genotoxic. The compound was found to be devoid of 
mutagenic activity (with and without metabolic activation) in 
salmonella typhimurium and escherichia coli (Ames assay). Acetamiprid 
was also, not mutagenic in an in vitro mammalian cell gene mutation 
assay on Chinese hamster ovary (CHO) cells (HPRT locus, with and 
without metabolic activation). Acetamiprid did not induce unscheduled 
DNA synthesis (UDS) in either rat liver primary cell cultures or in 
mammalian liver cells in vivo. In an in vitro, chromosomal aberration 
study using CHO cells, acetamiprid was positive when tested under 
metabolic activation at cytotoxic dose levels; no effect was detected 
without metabolic activation. Acetamiprid was non-clastogenic in an in 
vivo chromosomal aberration study in rat bone marrow. It was negative 
also, in an in vivo mouse bone marrow micronucleus assay.
    3. Reproductive and developmental toxicity. In the multi-generation 
rat reproduction study a no observed effect level (NOEL) of 100 ppm was 
established based on decreased body weight gains and a reproduction 
NOEL of 800 ppm (highest dose tested) was established for reproductive 
performance and fertility. In the rat teratology study the 
developmental NOEL was 50 milligrams/kilogram/day (mg/kg/day) (maternal 
NOEL of 16 mg/kg/day based on decreased body weight and food 
consumption) and in the rabbit teratology study the developmental NOEL 
was 30 mg/kg/day (maternal NOEL of 15 mg/kg/day based on decreased body 
weight and food consumption). In both the rat and rabbit studies there 
were no fetotoxic or teratogenic findings.
    4. Subchronic toxicity. In the 3-month dog feeding study a NOEL of 
800 ppm (32 mg/kg/day for both males and females) was established based 
on growth retardation and decreased food consumption.
     In the 3-month rat feeding study a NOEL of 200 ppm (12.4 and 14.6 
mg/kg/day respectively for male and female rats) was established based 
on liver cell hypertrophy at a dose of 800 ppm.
     In the 3-month mouse feeding study a NOEL of 400 ppm (53.2 and 
64.6 mg/kg/day respectively for male and female mice) was established 
based on increased liver/body weight ratio and decreased cholesterol in 
females at 800 ppm.
     A 13-week dietary neurotoxicity study for acetamiprid established 
a NOEL of 200 ppm (14.8 and 16.3 mg/kg for male and female rats) based 
on reduced body weight and food consumption decreases at 800 ppm. There 
was no evidence of neurotoxicity.
     A 21-day dermal study in rabbits at dose levels up to 1,000 mg/kg/
day caused no systemic toxicity, dermal irritation or 
histomorphological lesions in either sex tested.
    5. Chronic toxicity. In the 1-year dog study, the NOEL was 
established at 600 ppm (20 and 21 mg/kg/day for male and female dogs, 
respectively) based on growth retardation and decreased food 
consumption at a dose of 1,500 ppm.
     In the 18-month mouse study the NOEL was established at 130 ppm 
(20.3 and 25.2 mg/kg/day for male and female mice) based on growth 
retardation and hepatic toxicity at 400 ppm.
     In the 2-year rat study the NOEL was 160 ppm (7.1 and 8.8 mg/kg/
day for male and female rats) based on growth retardation and hepatic 
toxicity. There were no indications of carcinogenicity in either the 
rat or mouse chronic studies.
    6. Animal metabolism. The metabolism of acetamiprid is well 
understood and the primary animal metabolite is IM-2-1.
    7. Metabolite toxicology. Testing of IM-2-1 demonstrated that it is 
significantly less toxic than the parent acetamiprid and it is not 
being considered as part of the total toxic residue, therefore, no 
tolerance is being requested by the registrant. The acute oral 
LD50 of IM-2-1 is 2,543 mg/kg for male rats and 1,762 mg/kg 
for female rats.
    8. Endocrine disruption. Acetamiprid does not belong to a class of 
chemicals known or suspected of having adverse effects on the endocrine 
system. Developmental toxicity studies in rats and rabbits and a 
reproductive study in rats gave no indication that acetamiprid has any 
effects on endocrine function. The chronic feeding studies also, did 
not show any long-term effects related to endocrine systems.

C. Aggregate Exposure

    1. Dietary exposure. Acute and chronic dietary analyses were 
conducted to estimate exposure to potential acetamiprid residues in/on 
the following crops: Cole crop group, citrus crop group, fruiting 
vegetable crop group, pome fruit crop group, grapes, leafy vegetables, 
canola oil, mustard seed, cotton, and the tuberous and corm vegetable 
crop group using the Dietary Exposure Evaluation Model 
(DEEMTM) software. Exposure estimates to water were made 
based upon modeling.
    2. Food. The acute dietary exposure estimates at the 
99.9th percentile for the U.S. population was calculated to 
be 5.9% of the acute RfD. The population subgroup with the highest 
exposure was non-nursing infants at 15.4% of the acute RfD. The acute 
RfD was based on the NOEL of 10 mg/kg in the acute neurotoxicity study. 
Chronic dietary exposure estimates from residues of acetamiprid for the 
U.S. population was 0.3% of the chronic Populaton Adjusted Dose (cPAD). 
The subpopulation with the highest exposure was children 1-6 with 1.3% 
of the cPAD used. These values are based on projected percentages for 
percent of crop treated and field trial residues at maximum label rates 
and minimum PHI's with no reduction factors for common washing, 
cooking, or preparation practices. These can be considered conservative 
values. The cPAD was based on the NOEL of 7.1 mg/kg/day in the chronic 
rat study, an uncertainty factor of 100 to account for interspecies and 
intraspecies variations, and an FQPA safety factor of 3.
    3. Drinking water. EPA's standard operating procedure (SOP) for 
drinking water exposure and risk assessments was used to perform the 
drinking water analysis for acetamiprid. This SOP utilizes a variety of 
tools to conduct drinking water assessment. These tools include water 
models such as screening concentration in ground water (SCI-GROW), 
generic expected environmental concentration (GENEEC), pesticide root 
zone model/exposure

[[Page 47149]]

analysis modeling system (PRZM/EXAMS), and monitoring data. If 
monitoring data are not available then the models are used to predict 
potential residues in surface water and ground water. In the case of 
acetamiprid, monitoring data do not exist, therefore, GENEEC and 
SCIGROW models were used to estimate a water residue. The calculated 
drinking water levels of comparison (DWLOC) for acute and chronic 
exposures for all adults and children greatly exceed the modeled 
acetamiprid water residues, drinking water estimated concentrations 
(DWEC). The acute DWLOC values are 3,360 ppb for adults and 940 parts 
per billion (ppb) for children. The worst case DWEC for acute scenarios 
is calculated to be 13.27 ppb using the GENEEC surface water model. The 
chronic DWLOC values are 2,450 ppb for adults and 700 ppb for children. 
The DWEC for the worst case chronic scenario is 1.59 ppb (GENEEC).
    4. Non-dietary exposure. A ready to use, dilute formulation of 
acetamiprid is registered for insect control on outdoor ornamentals, 
vegetables and fruit trees. Based on surrogate exposure data obtained 
from a carbaryl study, the homeowner margin of exposure (MOE) was 
calculated to exceed ten million. Postapplication exposure resulting 
from contact with acetamiprid treated foliage resulted in an MOE in 
excess of 500,000.

D. Cumulative Effects

     EPA and ILSI are developing the methodologies to resolve the 
complex scientific issues concerning common mechanism of toxicity and 
how to cumulate pesticides in a quantitative manner. A determination 
has not been made that acetamiprid has a common mechanism of toxicity 
with other substances. Acetamiprid does not appear to produce a common 
toxic metabolite with other substances. A cumulative risk assessment 
was, therefore, not performed for this analysis.

E. Safety Determination

    1. U.S. population. Using the conservative assumptions described 
above, based on the completeness and reliability of the toxicity data, 
it is concluded that aggregate exposure to the proposed uses of 
acetamiprid will utilize at most 5.9% of the acute reference dose for 
the U.S. population, and is likely to be much less, as more realistic 
data and models are developed. EPA generally has no concern for 
exposures below 100% of the RfD because the RfD represents the level at 
or below which daily aggregate exposure over a lifetime will not pose 
appreciable risks to human health. Drinking water levels of comparison 
based on this exposure are much greater than conservative estimated 
concentrations, and would be expected to be well below the 100% level, 
if they occur at all. Therefore, there is a reasonable certainty that 
no harm will occur to the U.S. population from aggregate exposure to 
acetamiprid.
    2. Infants and children. In multi-generation reproduction and 
teratology studies, no adverse effects on reproduction were observed in 
either rats or rabbits. In the long term feeding studies in rats and 
mice there was no evidence of carcinogenicity. Acetamiprid was not 
mutagenic under the conditions of testing. Using the conservative 
exposure assumptions described in the exposure section above, the 
percent of the reference dose that will be used for short term 
aggregate exposure to residues of acetamiprid will be 15.4% for non-
nursing infants (the most highly exposed sub-group). This value is 
based on dietary exposure alone as only children over 7 are expected to 
have residential post-application exposure for the proposed acetamiprid 
uses. As in the adult situation, drinking water levels of comparison 
are much higher than the worst case drinking water estimated 
concentrations and would be expected to use well below 100% of the RfD, 
if they occur at all. Therefore, there is a reasonable certainty that 
no harm will occur to infants and children from aggregate exposure to 
residues of acetamiprid.

F. International Tolerances

     Acetamiprid is registered for use on food crops in several 
countries outside the United States (e.g., maximum residue levels 
(MRLs) are established in Canada and Japan).
[FR Doc. 04-17507 Filed 8-3-04; 8:45 am]
BILLING CODE 6560-50-S