[Federal Register Volume 65, Number 61 (Wednesday, March 29, 2000)]
[Notices]
[Pages 16598-16602]
From the Federal Register Online via the Government Publishing Office [www.gpo.gov]
[FR Doc No: 00-7742]


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ENVIRONMENTAL PROTECTION AGENCY

[PF-925; FRL-6496-9]


Notice of Filing a Pesticide Petition to Establish a Tolerance 
for Certain Pesticide Chemicals in or on Food

AGENCY: Environmental Protection Agency (EPA).

ACTION: Notice.

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SUMMARY: This notice announces the initial filing of a pesticide 
petition proposing the establishment of regulations for residues of a 
certain pesticide chemical in or on various food commodities.

DATES: Comments, identified by docket control number PF-925, must be 
received on or before April 28, 2000.

ADDRESSES: Comments may be submitted by mail, electronically, or in 
person. Please follow the detailed instructions for each method as 
provided in Unit I.C. of the SUPPLEMENTARY INFORMATION. To ensure 
proper receipt by EPA, it is imperative that you identify docket 
control number PF-925 in the subject line on the first page of your 
response.

FOR FURTHER INFORMATION CONTACT:  By mail: James Tompkins, Registration 
Support Branch, Registration Division (7505W), Office of Pesticide 
Programs, Environmental Protection Agency, Ariel Rios Bldg., 1200 
Pennsylvania Ave., NW., Washington, DC 20460; telephone number: (703) 
305-5697; e-mail address: [email protected].

SUPPLEMENTARY INFORMATION:

I. General Information

A. Does this Action Apply to Me?

    You may be affected by this action if you are an agricultural 
producer, food manufacturer or pesticide manufacturer. Potentially 
affected categories and entities may include, but are not limited to:

 
------------------------------------------------------------------------
                                                          Examples of
           Categories                 NAICS codes         potentially
                                                       affected entities
------------------------------------------------------------------------
Industry                          111                 Crop production
                                  112                 Animal production
                                  311                 Food manufacturing
                                  32532               Pesticide
                                                       manufacturing
------------------------------------------------------------------------

    This listing is not intended to be exhaustive, but rather provides 
a guide for readers regarding entities likely to be affected by this 
action. Other types of entities not listed in the table could also be 
affected. The North American

[[Page 16599]]

Industrial Classification System (NAICS) codes have been provided to 
assist you and others in determining whether or not this action might 
apply to certain entities. If you have questions regarding the 
applicability of this action to a particular entity, consult the person 
listed under FOR FURTHER INFORMATION CONTACT.

B. How Can I Get Additional Information, Including Copies of this 
Document and Other Related Documents?

    1. Electronically. You may obtain electronic copies of this 
document, and certain other related documents that might be available 
electronically, from the EPA Internet Home Page at http://www.epa.gov/. 
To access this document, on the Home Page select ``Laws and 
Regulations'' and then look up the entry for this document under the 
``Federal Register--Environmental Documents.'' You can also go directly 
to the Federal Register listings at http://www.epa.gov/fedrgstr/.
    2. In person. The Agency has established an official record for 
this action under docket control number PF-925. The official record 
consists of the documents specifically referenced in this action, any 
public comments received during an applicable comment period, and other 
information related to this action, including any information claimed 
as confidential business information (CBI). This official record 
includes the documents that are physically located in the docket, as 
well as the documents that are referenced in those documents. The 
public version of the official record does not include any information 
claimed as CBI. The public version of the official record, which 
includes printed, paper versions of any electronic comments submitted 
during an applicable comment period, is available for inspection in the 
Public Information and Records Integrity Branch (PIRIB), Rm. 119, 
Crystal Mall #2, 1921 Jefferson Davis Highway, Arlington, VA, from 8:30 
a.m. to 4 p.m., Monday through Friday, excluding legal holidays. The 
PIRIB telephone number is (703) 305-5805.

C. How and to Whom Do I Submit Comments?

    You may submit comments through the mail, in person, or 
electronically. To ensure proper receipt by EPA, it is imperative that 
you identify docket control number PF-925 in the subject line on the 
first page of your response.
    1. By mail. Submit your comments to: Public Information and Records 
Integrity Branch (PIRIB), Information Resources and Services Division 
(7502C), Office of Pesticide Programs (OPP), Environmental Protection 
Agency, Ariel Rios Bldg., 1200 Pennsylvania Ave., NW., Washington, DC 
20460.
    2. In person or by courier. Deliver your comments to: Public 
Information and Records Integrity Branch (PIRIB), Information Resources 
and Services Division (7502C), Office of Pesticide Programs (OPP), 
Environmental Protection Agency, Rm. 119, Crystal Mall #2, 1921 
Jefferson Davis Highway, Arlington, VA. The PIRIB is open from 8:30 
a.m. to 4 p.m., Monday through Friday, excluding legal holidays. The 
PIRIB telephone number is (703) 305-5697.
    3. Electronically. You may submit your comments electronically by 
e-mail to: ``[email protected],'' or you can submit a computer disk as 
described above. Do not submit any information electronically that you 
consider to be CBI. Avoid the use of special characters and any form of 
encryption. Electronic submissions will be accepted in Wordperfect 6.1/
8.0 or ASCII file format. All comments in electronic form must be 
identified by docket control number PF-925. Electronic comments may 
also be filed online at many Federal Depository Libraries.

D. How Should I Handle CBI That I Want to Submit to the Agency?

    Do not submit any information electronically that you consider to 
be CBI. You may claim information that you submit to EPA in response to 
this document as CBI by marking any part or all of that information as 
CBI. Information so marked will not be disclosed except in accordance 
with procedures set forth in 40 CFR part 2. In addition to one complete 
version of the comment that includes any information claimed as CBI, a 
copy of the comment that does not contain the information claimed as 
CBI must be submitted for inclusion in the public version of the 
official record. Information not marked confidential will be included 
in the public version of the official record without prior notice. If 
you have any questions about CBI or the procedures for claiming CBI, 
please consult the person identified under FOR FURTHER INFORMATION 
CONTACT.

E. What Should I Consider as I Prepare My Comments for EPA?

    You may find the following suggestions helpful for preparing your 
comments:
    1. Explain your views as clearly as possible.
    2. Describe any assumptions that you used.
    3. Provide copies of any technical information and/or data you used 
that support your views.
    4. If you estimate potential burden or costs, explain how you 
arrived at the estimate that you provide.
    5. Provide specific examples to illustrate your concerns.
    6. Make sure to submit your comments by the deadline in this 
notice.
    7. To ensure proper receipt by EPA, be sure to identify the docket 
control number assigned to this action in the subject line on the first 
page of your response. You may also provide the name, date, and Federal 
Register citation.

II. What Action is the Agency Taking?

    EPA has received a pesticide petition as follows proposing the 
establishment and/or amendment of regulations for residues of a certain 
pesticide chemical in or on various food commodities under section 408 
of the Federal Food, Drug, and Cosmetic Act (FFDCA), 21 U.S.C. 346a. 
EPA has determined that this petition contains data or information 
regarding the elements set forth in section 408(d)(2); however, EPA has 
not fully evaluated the sufficiency of the submitted data at this time 
or whether the data supports granting of the petition. Additional data 
may be needed before EPA rules on the petition.

List of Subjects

    Environmental protection, Agricultural commodities, Feed additives, 
Food additives, Pesticides and pests, Reporting and recordkeeping 
requirements.

    Dated: March 21, 2000.
James Jones,
Director, Registration Division, Office of Pesticide Programs.

Summary of Petition

    The petitioner summary of the pesticide petition is printed below 
as required by section 408(d)(3) of the FFDCA. The summary of the 
petition was prepared by the petitioner and represents the view of the 
petitioners. EPA is publishing the petition summary verbatim without 
editing it in any way. The petition summary announces the availability 
of a description of the analytical methods available to EPA for the 
detection and measurement of the pesticide chemical residues or an 
explanation of why no such method is needed.

[[Page 16600]]

Bayer Corporation

0F6095

    EPA has received a pesticide petition (0F6095) from Bayer 
Corporation, 8400 Hawthorn Road, Kansas City, MO 64120-0013 proposing, 
pursuant to section 408(d) of the Federal Food, Drug, and Cosmetic Act 
(FFDCA), 21 U.S.C. 346a(d), to amend 40 CFR 180.527 by establishing a 
tolerance for residues of flufenacet, N-(4-fluorophenyl)-N-(1-
methylethyl)-2-5-(trifluoromethyl)-1,3,4-thiadiazol-2-yl oxyacetamide 
and metabolites containing the 4-fluoro-N-methylethyl benzenamine 
moiety in or on the raw agricultural commodities (RAC) wheat grain, 
wheat forage, wheat hay, wheat bran, wheat germ, wheat straw, seed-
grass forage, seed-grass forage from re-growth, seed-grass hay from re-
growth, seed-grass straw, sweet corn kernel plus cob with husks removed 
at 0.5, 9.0, 1.0, 1.0, 0.5, 0.5, 18.0, 0.1, 0.5, 0.5, 0.05 parts per 
million (ppm), respectively. EPA has determined that the petition 
contains data or information regarding the elements set forth in 
section 408(d)(2) of the FFDCA; however, EPA has not fully evaluated 
the sufficiency of the submitted data at this time or whether the data 
supports granting of the petition. Additional data may be needed before 
EPA rules on the petition.

A. Residue Chemistry

    1. Plant metabolism. The nature of the residue in field corn, sweet 
corn, wheat, seed-grasses, soybeans, rotational crops, and livestock is 
adequately understood. The residues of concern for the tolerance 
expression are flufenacet parent and its metabolites containing the 4-
fluoro-N-methylethyl benzenamine moiety. Based on the results of animal 
metabolism studies it is unlikely that secondary residues would occur 
in animal commodities from the use of flufenacet on field corn, sweet 
corn, soybeans, wheat, and seed-grasses.
    2. Analytical method. An adequate analytical method, gas 
chromatography/mass spectrometry (GC/MS) with selected ion monitoring, 
is available for enforcement purposes. Because of the long lead time 
from establishing these tolerances to publication of the enforcement 
methodology in the Pesticide Analytical Manual, Vol. II, the analytical 
methodology is being made available in the interim to anyone interested 
in pesticide enforcement when requested from: Calvin Furlow, Public 
Information and Records Integrity Branch, Information Resources and 
Services Division (7502C), Office of Pesticide Programs, Environmental 
Protection Agency, Ariel Rios Bldg., 1200 Pennsylvania Ave., NW., 
Washington, DC 20460; telephone number: (703-305-5937).
    3. Magnitude of residues. Time-limited tolerances exist for the 
combined residues of flufenacet, N-(4-fluorophenyl)-N-(1-methylethyl)-
2-5-(trifluoromethyl)-1,3,4-thiadiazol-2-yloxyacetamide and metabolites 
containing the 4-fluoro-N-methylethyl benzenamine moiety in or on field 
corn grain at 0.05 ppm, field corn forage at 0.4 ppm, field corn stover 
at 0.4 ppm, soybean seed at 0.1 ppm, alfalfa forage at 0.1 ppm, alfalfa 
hay at 0.1 ppm, alfalfa seed at 0.1 ppm, clover forage at 0.1 ppm, 
clover hay at 0.1 ppm, Crop Group 15 (cereal grains) at 0.1 ppm, Crop 
Group 16 (forage, stover and hay of cereal grains) at 0.1 ppm, and 
Group 17 (grass forage and grass hay) at 0.1 ppm.

B. Toxicological Profile

    1. Acute toxicity--i. Technical grade flufenacet has a low to 
moderate order of toxicity in rats by the oral route of exposure. The 
acute oral LD50 was 1,617 milligrams/kilograms (mg/kg) for 
males and 589 mg/kg for females.
    ii. A dermal toxicity study on technical grade flufenacet revealed 
low acute toxicity to rats. The dermal LD50 for both sexes 
was > 2,000 mg/kg, the highest dose tested (HDT).
    iii. An acute inhalation study on technical grade flufenacet showed 
low toxicity in rats with a 4-hour liquid aerosol LC50 for 
males and females of > 3,740 mg/m3 air, the highest 
concentration tested.
    iv. An eye irritation study on technical grade flufenacet in 
rabbits showed minimal irritation to the conjunctiva completely 
reversible within 7 days.
    v. A dermal irritation study on technical grade flufenacet in 
rabbits did not produced any irritation.
    vi. Skin sensitization studies on technical grade flufenacet in 
guinea pigs have produced equivocal results. A skin sensitization 
potential was exhibited under the conditions of a maximization test, 
whereby, there was no skin sensitization potential when tested by the 
Buehler topical closed patch technique.
    2. Genotoxicity. Flufenacet was negative for mutagenic/genotoxic 
effects in a Gene mutation/in vitro assay in bacteria, a Gene mutation/
in vitro assay in Chinese hamster (CH) lung fibroblasts cells, a 
Cytogenetics/in vitro assay in CH ovary cells, a Cytogenetics/in vivo 
mouse micronucleus assay, and an in vitro unscheduled DNA synthesis 
assay in primary rat hepatocytes.
    3. Reproductive and developmental toxicity--i. A 2-generation rat 
reproduction study with a parental systemic no observed adverse effect 
level (NOAEL) of 20 ppm (1.4 mg/kg/day in males and 1.5 mg/kg/day in 
females) and a reproductive NOAEL of 20 ppm (1.3 mg/kg/day) and a 
parental systemic lowest observed adverse effect level (LOAEL) of 100 
ppm (7.4 mg/kg/day in males and 8.2 mg/kg/day in females), based on 
increased liver weight in F1 females and hepatocytomegaly in 
F1 males, and a reproductive LOAEL of 100 ppm (6.9 mg/kg/
day) based on increased pup death in early lactation (including 
cannibalism) for F1 litters and the same effects in both 
F1 and F2 pups at the high dose level of 500 ppm 
(37.2 mg/kg/day in males and 41.5 mg/kg/day in females), respectively.
    ii. A rat developmental study with a maternal NOAEL of 25 mg/kg/day 
and with a maternal LOAEL of 125 mg/kg/day based on decreased body 
weight gain initially and a developmental NOAEL of 25 mg/kg/day and a 
developmental LOAEL of 125 mg/kg/day based on decreased fetal body 
weight, delayed development mainly delays in ossification in the skull, 
vertebrae, sternebrae, and appendages, and an increase in the incidence 
of extra ribs.
    iii. A rabbit developmental study with a maternal NOAEL of 5 mg/kg/
day and a maternal LOAEL of 25 mg/kg/day based on histopathological 
finds in the liver and a developmental NOAEL of 25 mg/kg/day and a 
developmental LOAEL of 125 mg/kg/day based on increased skeletal 
variations.
    4. Subchronic toxicity--i. A 84-day rat feeding study with a NOAEL 
less than 100 ppm (6.0 mg/kg/day) for males and a NOAEL of 100 ppm (7.2 
mg/kg/day) for females and with a LOAEL of 100 ppm (6.8 mg/kg/day) for 
males based on suppression of thyroxine (T4) level, and a LOAEL of 400 
ppm (28.8 mg/kg/day) for females based on hematology, and clinical 
chemistry findings.
    ii. A 13-week mouse feeding study with a NOAEL of 100 ppm (18.2 mg/
kg/day for males and 24.5 mg/kg/day for females), and a LOAEL of 400 
ppm (64.2 mg/kg/day for males and 91.3 mg/kg/day for females) based on 
histopathology of the liver, spleen and thyroid.
    iii. A 13-week dog dietary study with a NOAEL of 50 ppm (1.70 mg/
kg/day for males and 1.67 mg/kg/day for females), and a LOAEL of 200 
ppm (6.90 mg/kg/day for males and 7.20 mg/kg/day for females), based on 
evidence that the bio-transformation capacity of the liver has

[[Page 16601]]

been exceeded (as indicated by increase in LDH, liver weight, ALK and 
hepatomegaly), globulin and spleen pigment in females, decreased T4 and 
ALT values in both sexes, decreased albumin in males, and decreased 
serum glucose in females.
    iv. A 21-day rabbit dermal study with the dermal irritation NOAEL 
of 1,000 mg/kg/day for males and females, and a systemic NOAEL of 20 
mg/kg/day for males and 150 mg/kg/day for females, and a systemic LOAEL 
of 150 mg/kg/day for males and 1,000 mg/kg/day for females based on 
clinical chemistry data (decreased T4 and FT4 levels in both sexes) and 
centrilobular hepatocytomegaly in females.
    5. Chronic toxicity--i. A 1-year dog chronic feeding study with a 
NOAEL was 40 ppm (1.29 mg/kg/day in males and 1.14 mg/kg/day in 
females), and a LOAEL of 800 ppm (27.75 mg/kg/day in males and 26.82 
mg/kg/day in females) based on increased alkaline phosphatase, kidney, 
and liver weight in both sexes, increased cholesterol in males, 
decreased T2, T4 and ALT values in both sexes, and increased incidences 
of microscopic lesions in the brain, eye, kidney, spinal cord, sciatic 
nerve, and liver.
    ii. A rat chronic feeding/carcinogenicity study with a NOAEL less 
than 25 ppm (1.2 mg/kg/day in males and 1.5 mg/kg/day in females), and 
a LOAEL of 25 ppm (1.2 mg/kg/day in males and 1.5 mg/kg/day in females) 
based on methemoglobinemia, and multi-organ effects in blood, kidney, 
spleen, heart, and uterus. Under experimental conditions the treatment 
did not alter the spontaneous tumor profile.
    iii. In a mouse carcinogenicity study the NOAEL was less than 50 
ppm (7.4 mg/kg/day) for males and the NOAEL was 50 ppm (9.4 mg/kg/day) 
for females. The LOAEL was 50 ppm (7.4 mg/kg/day) for males and the 
LOAEL was 200 ppm (38.4 mg/kg/day) for females based on cataract 
incidence and severity. There was no evidence of carcinogenicity for 
flufenacet in this study.
    6. Animal metabolism. A rat metabolism study showed that radio-
labeled flufenacet was rapidly absorbed and metabolized by both sexes. 
Urine was the major route of excretion at all dose levels and smaller 
amounts were excreted via the feces.
    7. Metabolite toxicology. A 55-day dog study with subcutaneous 
administration of thiadone flufenacet metabolite supports the 
hypothesis that limitations in glutathione interdependent pathways and 
antioxidant stress result in metabolic lesions in the brain and heart 
following flufenacet exposure.
    8. Endocrine disruption. EPA is required to develop a screening 
program to determine whether certain substances (including all 
pesticides and inerts) may have an effect in humans that is similar to 
an effect produced by a naturally occurring estrogen, or such other 
effect. The Agency is currently working with interested stakeholders, 
including other government agencies, public interest groups, industry 
and research scientists in developing a screening and testing program 
and a priority setting scheme to implement this program. Congress has 
allowed 3 years from the passage of FQPA (August 3, 1999) to implement 
this program. At that time, EPA may require further testing of this 
active ingredient and end use products for endocrine disrupter effects. 
Based on the toxicological findings for flufenacet relating to 
endocrine disruption effects, flufenacet should be considered as a 
candidate for evaluation as an endocrine disrupter when the criteria 
are established.
    9. Other studies--i. An acute rat neurotoxicity study with a NOAEL 
less than 75 mg/kg/day and a LOAEL of 75 mg/kg/day based on decreased 
motor activity in males.
    ii. A rat subchronic neurotoxicity study with a NOAEL of 120 ppm 
(7.3 mg/kg/day in males and 8.4 mg/kg/day in females), and a LOAEL of 
600 ppm (38.1 mg/kg/day in males and 42.6 mg/kg/day in females) based 
on microscopic lesions in the cerebellum/medulla and spinal cords.

C. Aggregate Exposure

    1. Dietary exposure--i. Food. Dietary exposure to residues of a 
pesticide in a food commodity are estimated by multiplying the average 
daily consumption of the food forms of that commodity by the tolerance 
level or the anticipated pesticide residue level. In evaluating food 
exposures, varying consumption patterns of major identifiable subgroups 
of consumers, including infants and children is taken into account. A 
refined dietary risk assessment was performed and adjustments were made 
to account for market share and processing factors. The residues in the 
diet (food only) are calculated to be 0.000078 mg/kg bwt day or 1.9% of 
the RfD for the general U.S. population and 0.000174 mg/kg bwt day or 
4.4% of the RfD for non-nursing infants (> 1-year)
    ii. Drinking water. Residues of flufenacet in drinking water may 
comprise up to 0.0039 mg/kg bwt day (0.0040-0.000078 mg/kg bwt day) for 
the U.S. population and 0.0038 mg/kg bwt day (0.00400-0.000174 mg/kg 
bwt day) for children 1-6 years old.
    The drinking water levels of concern (DWLOCs) for chronic exposure 
to flufenacet in drinking water calculated for the U.S. population was 
136 parts per billion (ppb) assuming that an adult weighs 70 kg and 
consumes a maximum of 2 liters of water per day. For children (1-6 
years old), the DWLOC was 37.7 ppb assuming that a child weighs 10 kg 
and consumes a maximum of 1 liter of water per day.
    The drinking water estimated concentration (DWECs) for ground water 
(parent flufenacet and degradate thiadone) calculated from the 
monitoring data is 0.03 ppb for chronic concentrations which does not 
exceed DWLOC of 37.7 ppb for children (1-6 years old). The DWEC for 
surface water based on the computer models PRZM 2.3 and EXAMS 2.97.5 
was calculated to be 14.2 ppb for chronic concentration (parent 
flufenacet and degradate thiadone) which does not exceed the DWLOC of 
37.7 ppb for children (1-6 years old).
    2. Non-dietary exposure. There are no non-food uses of flufenacet 
currently registered under the Federal Insecticide, Fungicide, and 
Rodenticide Act, as amended. No non-dietary exposures are expected for 
the general population.

D. Cumulative Effects

    Flufenacet is structurally a thiadiazole. EPA is not aware of any 
other pesticides with this structure. For flufenacet, EPA has not yet 
conducted a detailed review of common mechanisms to determine whether 
it is appropriate, or how to include this chemical in a cumulative risk 
assessment. After EPA develops a methodology to address common 
mechanism of toxicity issues to risk assessments, the Agency will 
develop a process (either as part of the periodic review of pesticides 
or otherwise) to reexamine these tolerance decisions. Unlike other 
pesticides for which EPA has followed a cumulative risk approach based 
on a common mechanism of toxicity, flufenacet does not appear to 
produce a toxic metabolite produced by other substances. For the 
purposes of these tolerance actions, therefore, EPA has not assumed 
that flufenacet has a common mechanism of toxicity with other 
substances.

E. Safety Determination

    1. U.S. population. As presented previously, the exposure of the 
U.S. general population to flufenacet is low, and the risks, based on 
comparisons to the RfD, are minimal. The margins of safety from the use 
of flufenacet are within EPA's acceptable limits. Bayer

[[Page 16602]]

Corporation concludes that there is a reasonable certainty that no harm 
will result to the U.S. population from aggregate exposure to 
flufenacet residues.
    2. Infants and children. In assessing the potential for additional 
sensitivity of infants and children to residues of flufenacet, EPA 
considered data from developmental toxicity studies in the rat and 
rabbit and a 2-generation reproduction study in the rat. The 
developmental toxicity studies are designed to evaluate adverse effects 
on the developing organism resulting from pesticide exposure during 
prenatal development to one or both parents. Reproduction studies 
provide information relating to effects from exposure to the pesticide 
on the reproductive capability of mating animals and data on systemic 
toxicity. FFDCA section 408 provides that EPA shall apply an additional 
tenfold margin of safety for infants and children in the case of 
threshold effects to account for prenatal and postnatal toxicity and 
the completeness of the data base unless EPA determines that a 
different margin of safety will be safe for infants and children. 
Although there is no indication of increased sensitivity to young rats 
or rabbits following prenatal and/or postnatal exposure to flufenacet 
in the standard developmental and reproductive toxicity studies, an 
additional developmental neurotoxicity study, which is not normally 
required, is needed to access the susceptibility of the offspring in 
function/neurological development. Therefore, EPA has required that a 
developmental neurotoxicity study be conducted with flufenacet and a 
threefold safety factor for children and infants will be used in the 
aggregate dietary acute and chronic risk assessment. Although there is 
no indication of additional sensitivity to young rats or rabbits 
following prenatal and/or postnatal exposure to flufenacet in the 
developmental and reproductive toxicity studies; the Agency concluded 
that the FQPA safety factor should not be removed but instead reduced 
because: (i) There was no assessment of susceptibility of the offspring 
in functional/neurological developmental and reproductive studies; (ii) 
there is evidence of neurotoxicity in mice, rats, and dogs; (iii) there 
is concern for thyroid hormone disruption.

F. International Tolerances

    Maximum residue levels are established or proposed for countries of 
the European Communities in the following commodities: cereals at 0.5 
ppm, corn at 0.5 ppm, potato at 0.1 ppm, sunflower at 0.05 ppm, soybean 
at 0.05 ppm, animal meat at 0.05 ppm, animal edible offal's at 0.05 
ppm, animal fat at 0.05 ppm, milk at 0.01 ppm, and eggs at 0.05 ppm.
[FR Doc. 00-7742 Filed 3-28-00; 8:45 am]
BILLING CODE 6560-50-F